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A cancer cell-intrinsic GOT2-PPARδ axis suppresses antitumor immunity | bioRxiv
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Bi-allelic GOT2 Mutations Cause a Treatable Malate-Aspartate Shuttle-Related Encephalopathy - ScienceDirect
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A cancer cell-intrinsic GOT2-PPARδ axis suppresses antitumor immunity | bioRxiv
SIRT3‐dependent GOT2 acetylation status affects the malate–aspartate NADH shuttle activity and pancreatic tumor growth | The EMBO Journal
The malate-aspartate NADH shuttle (MAS) as the major redox shuttle in... | Download Scientific Diagram
Preventing BRCA1/ZBRK1 repressor complex binding to the GOT2 promoter results in accelerated aspartate biosynthesis and promotion of cell proliferation - Hong - 2019 - Molecular Oncology - Wiley Online Library